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CMA Chlormadinone Acetate Progesterone CAS 302-22-7 for Emergency Contraception

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CMA Chlormadinone Acetate Progesterone CAS 302-22-7 for Emergency Contraception

Brand Name : JCJ
Model Number : CAS: 302-22-7
Certification : GMP,ISO,SGS,BV,KOSHER
Place of Origin : China
MOQ : Negotiable
Price : Negotiable
Payment Terms : Western Union, MoneyGram, T/T, BitCoin
Supply Ability : Mass in stock
Delivery Time : 24hours after payment due
Packaging Details : Disguised package, foil bag, etc.(Negotiable)
Alias : Chlormadinone Acetate;Progesterone;CMA
Appearance : white· Powder
Usage : for Emergency Contraception
Policy : Re-Shipping Policy
EINECS : 206-118-0
Package : foil bag
Shipment : EMS, FedEx, TNT, DHL, UPS
Service : 7/24
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CMA Chlormadinone Acetate Progesterone CAS 302-22-7 for Emergency Contraception


Product Name:Chlormadinone acetate
CAS NO.: 302-22-7
EINECS:206-118-0
Assay: 99.2%
Molecular Formula: C23H29ClO4
Molecular Weight:404.93
Specification: CP2000, JP14
Packing:foil bags
Delivery: Within 24 hours after payment.

MOQ :10 grams.
Appearance:yellow crystalline powder



TEST ITEMS

SPECIFICATION

RESULTS

Description

White or Almost White Crystalline Powder

white powder

Identification

A.B.

Positive

Assay

97.0~102.0%

99.3%

Specific Rotation

+101°~+105°

+102.6°

Loss On Drying

1.0%max

0.27%

Melting Point

153~157°C

153.0~155.0°C

Organic Volatile Impurities

meets the requirement.

Conforms

Related Substances

meets the requirement.

Conforms

Residual Solvents

meets the requirement.

Conforms

Particle Size

100% ≤ 20 microns

Conforms

Conclusion

The specification conform with USP30 standard

Product Description :

(1)Chlormadinone acetate (CMA) is a derivative of progesterone (17-acetoxy-6-chloro-4,6-pregnadiene-3,20-dione), first synthesized in 1961 and is used as an orally effective progestogen in hormone replacement therapy (HRT), and in combination with ethinyl estradiol (EE) in contraception since 1999.
(2)Chlormadinone acetate has a strong progestogenic effect about one-third higher than that of progesterone and may vary depending on the previous effect of an estrogen, i.e., estrogens may promote the formation of progesterone receptors and proliferation of the endometrium. Like progesterone, it is anti-estrogenic and has no partial androgenic effect (at the doses used for contraception and HRT).
(3)In contrast to progesterone, it has a slight glucocorticoid effect, a pronounced anti-androgenic effect and no anti-mineralocorticoid effect. No pregnancy-maintaining effect of CMA has been demonstrated in humans.
(4)The anti-androgenic effect of CMA is presumed to be the result of both its binding to androgen receptors competitively inhibiting the effect of endogenous testosterone and dihydrotestosterone and the competitive inhibition of 5-reductase. In this respect, dosing of CMA is crucial; agonistic effects are observed when doses are increased from those optimal for an antagonistic effect.
(5)Chlormadinone acetate has a strong anti-gonadotropic effect, through negative feedback on gonadotropin secretion, and has been used for more than 20 years alone for contraception in arterial risk patients. The clinical and metabolic tolerability of CMA has been demonstrated in numerous clinical studies with duration of treatment of up to 2.5 years.
(6)The more recent application of CMA as an oral contraceptive in combination with EE (Neo Eunomin, Belara) has proven highly successful, with studies reporting excellent contraceptive efficacy, high tolerability and adherence due to a good side effect profile and positive effects on preexisting dysmenorrhea, skin and hair conditions.

Usage:

Chlormadinone acetate (CMA) is a derivative of naturally secreted progesterone that shows high affinity and activity at the progesterone receptor. It has an anti-estrogenic effect and, in contrast to natural progesterone, shows moderate anti-androgenic properties.

CMA acts by blocking androgen receptors in target organs and by reducing the activity of skin 5alpha-reductase. It suppresses gonadotropin secretion and thereby reduces ovarian and adrenal androgen production.

CMA shows high contraceptive efficacy by inhibiting ovulation due to its ability to suppress or disrupt endogenous gonadotropin secretion and, by this, inhibits follicular growth and maturation. In addition, it suppresses endometrial thickness and increases the viscosity of cervical mucus.


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Product nameCAS
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Anastrozole120511-73-1
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BOLDENONE CYPIONATE/
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Testosterone phenylpropionate1255-49-8
Nandrolone phenylpropionate62-90-8
Estradiol benzoate50-50-0
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Nandrolone 17-propionate7207-92-3
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Estriol50-27-1
Testosterone acetate1045-69-8
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Methandienone (Dianabol)72-63-9
Dutasteride164656-23-9
vardenafil224785-91-5
Finasteride98319-26-7
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Formestane566-48-3
Testosterone58-22-0
Oestradiol 17-heptanoate4956-37-0
Testosterone enanthate315-37-7
Clomiphene50-41-9
Tamoxifen citrate54965-24-1
Testosterone decanoate5721-91-5
19-Nortestoterone decanoate360-70-3
Testosterone cypionate58-20-8
Nandrolone cypionate601-63-8
Testosterone Sustanon 250/
Testosterone Sustanon 100/
hydrochloride129938-20-1
Megestrol/
WIN 55,212-2 MESYLATE131543-23-2
17-Methyltestosterone58-18-4
Methylepitiostanol (Epistane)4267-80-5
Methasterone (superdrol)3381-88-2
Methyltrienolone965-93-5
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Oxymetholone434-07-1
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Methenolone enanthate303-42-4
Mibolerone/
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Misoprostol59122-46-2
Nandrolone434-22-0
Drostanolone Propionate521-12-0
Drostanolone Enanthate521-12-0
Dehydroisoandrosterone (DHEA)53-43-0
Ethinyl estradiol57-63-6
Ethisterone434-03-7
Trenbolone base/
Trenbolone acetate10161-34-9
Trenbolone Enanthate10161-33-8
Trenbolone cyclohexylmethylcarbonate23454-33-3
T36893-2-3
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Testosterone Undecanoate5949-44-0
Nandrolone undecylate/
Altrenogest850-52-2
Tamoxifen10540-29-1
Tibolone5630-53-5
Cialis171596-29-5
hydrochloride129938-20-1
Oxandrolone53-39-4
Eplerenone107724-20-9
Exemestane107868-30-4
Testosterone isocaproate15262-86-9
1-Testosterone1965-6-5
Mestanolone521-11-9
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